Consuming fewer calories has a protective effect against developing hepatocellular carcinoma ( primary liver cancer) associated with hepatitis C virus infection, and non alcoholic fatty liver disease, according to a rodent study published in the Journal liver cancer.
Editor’s Note: Recently, worldwide increases in obesity and metabolic syndrome have raised the prevalence of primary liver cancer derived from non alcoholic fatty liver disease (NAFLD) and non alcoholic steatohepatitis (NASH), Indicating a close relationship between overnutrition and liver tumorigenesis, the authors stated.
The study used mice with the liver cancer core gene that spontaneously developed fatty liver and tumors. For 15 months, the animals were given either a control diet that allowed them to eat as much as they liked, or a diet that contains 30% fewer calories than the controls.
At the end of 15 months, animals that received calorie restricted diets had fewer and smaller liver tumors, less liver oxidative stress, lower inflammation, downregulation of pro- cancer mediators, increased autophagy(cell self degradation), as well as other improvements, compared to the control group.
In the News: Lower Alzheimers’ Risk with Flavonols
What are Phytochemicals?
Phyto chemicals are biologically active substances in plants that have positive effects on health They are also called phyto- nutrients. They perform a variety of functions including these roles:
Antioxidants
Inhibitors of inflammation
Preventers of infectious disease
Flavonols act as antioxidants in the body and the good news about flavanols has made chocolate a health food. Cocoa, the main ingredient in chocolate is a rich source of flavonols.. Regular intake of flavonols such as a daily consumption of a cup of hot chocolate made with cocoa powder is related to improved blood flow, reduced blood pressure, and decreased risk of heart disease and stroke. Flavonols are also found in good amounts in foods such as berries, wine, and tea.
An article in the Journal Neurology reported an association between consuming more compounds known as flavanols, and a lower risk of developing Alzheimer’s disease. Flavanols are found in many fruits and vegetables as well as in tea and chocolate.
The study included 921 participants with an average age of 81. The subjects the subjects did not have Alzheimer’s disease at the beginning of the study. Questionnaires that were completed at enrollment and then annually during a six year average follow-up period, provided data on dietary intake that was analyzed for flavonol content. Participants were also evaluated yearly for the presence of Alzheimer’s disease. Over the course of follow up , 220 individuals developed the disease. Participants were then divided into five groups, according to their level of flavonol intake. Among those whose intake was highest, at an average of 15.3 milligrams per day, 15% developed Alzheimer’s disease , compared to 30% whose intake was lowest, at approximately 5.3 milligram per day.
The authors stated that “eating more fruits and vegetables and drinking more tea could be a fairly inexpensive and easy way for people to help stave off Alzheimer’s dementia.”
There is no real Mediterranean Diet – you know, the ones you read in diet books that offer set meal plans and do’s and don’ts with endless lists of breakfasts, lunches, dinners and of course, let’s not forget the snacks. (or maybe we should). The more recent habit of the American Diet is the increased eating opportunities fueled by the fact that we can eat just about anywhere and that mainly was the desire of big food companies (for more profits). But more about that in a future post.
“Over the span of more than eight decades, clinical research has continued to confirm that eating the Mediterranean way is a sound strategy for lifelong health. At its heart, The Mediterranean diet is precisely the way nutrition experts have been urging us to eat. It’s built on a foundation of whole, mostly unprocessed foods like whole grains, beans, and nuts. It embraces fruits and vegetables with abandon while being stingier with red meat and sweet treats. It includes moderate amounts of fish, eggs and dairy products though vegan and vegetarian versions are doable. It’s sprinkled with heart healthy vegetable oils , mostly olive oil, rather than saturated fat rich butter or lard and it can even include, if desired, a little alcohol traditionally red wine, enjoyed in moderation as part of a meal.”
“People who follow a Mediterranean diet also tend to live longer and perhaps age more gracefully. A recent meta analysis of four studies involving elderly patients found that those who adhered most closely to a Mediterranean style eating pattern had significantly less risk of becoming frail, an important measure of quality of life for older adults.”
The components of the diet are generally plant-based that give us ample vitamins and minerals, antioxidants, anti-inflammatory compounds, fiber, and good fats and carbohydrates. Long-term studies have shown lower rates of heart disease, diabetes, and a reduced risk of some cancers (breast, prostate, and colorectal).
“Encouraging research suggests that a Mediterranean pattern of eating may also have benefits for the brain. Several studies have linked the eating patterns to lower rates of depression, others notice a small but significantly lower risk of developing mild cognitive impairment and of progressing to Alzheimer’s disease.”
Source: Joyce Hendley. Mediterranean Diet: A delicious path to lifelong health. Eating Well Magazine.
Calorie restriction is not an easy thing to do in our obesigenic society. However, based on the the following study, even small changes seems to be able to result in not only weight loss but the beneficial effects on our overall health.
The debate about calories has continued for quite some time.
In 1918, Dr. Lulu Hunt Peters wrote the first diet book, “Diet and Health, With Key to the Calories.” The book was a best seller. She explained the new concept of calorie reduction for weight loss. To see her book CLICK HERE.
In 1958 Dr. Richard MacKarness published “Eat Fat and Grow Slim”. The title speaks for itself.
In 1971, Dr. Herman Taller wrote another best seller, “Calories Don’t Count”.
More recently, author Gary Taubes wrote a provocative book entitled “Good Calories, Bad Calories” and Nina Teicholz espoused the health benefits of calorie dense butter, meat and cheese, in her book called “The Big Fat Surprise.”
No wonder people are struggling with obesity and will continue to do so until we figure out the physiological, psychological, sociological and environmental complexities of weight gain, weight loss, and weight maintenance.
The following article emphasizes the health benefits of calorie restriction whether due to weight loss or the calorie deficit itself.
A new study found consuming a number of refined grains, such as croissants and white bread, is associated with a higher risk of major cardiovascular disease, stroke, and early death. The study was published in the British Medical Journal.
The study called the Prospective Urban Rural Epidemiology (PURE) study examined populations around the world involving 137, 130 participants in 21 countries and involved a diversity of low-, middle-, and high-income populations.
The results found that having more than seven servings of refined grains per day was associated with a 27% greater risk for early death, 33% greater risk for heart disease, and 47% greater risk for stroke. Those groups eating whole grains or white rice showed no significant adverse health effects.
Source: Simon Fraser University. “Eating more refined grains risk of heart attack, early death.” ScienceDaily, 19 February 2021. http://www.sciencedaily, com/releases/2021/02
According to David Sinclair, PhD in his recent book, Lifespan:Why We Age and Why We Don’t Have To, “Aging is a disease and that disease is treatable.” What if you could have some control over how many years you can live and live that life with reasonable health?
There is now a fascinating new surge in aging research and a lot of attention paid to the contribution of healthy lifestyles. According to Dr. Sinclair, professor of genetics at Harvard Medical School, the key is “activating newly discovered genes, the descendants of an ancient survival circuit that is both the cause of aging and the key to reversing it.” Sounds a little mystical? It makes a lot of sense (after reading the book). New ideas can change the way we think about aging and what we can do about it. Anyone who ages must read about these new concepts.
Spoiler Alert: From the article and study: Researchers estimated participants who didn’t follow any of the habits had a life expectancy at age 50 of 29 years for women and 25.5 years for men. Yet for those who did adopt these guidelines, after age 50 women could expect to live another 43.1 years, compared to 37.6 years on average for men. Ben Renner, Harvard Study: Adopting These 5 Healthy Habits can Add Decade To Your Life. Study Finds, June 16, 2018,
Intermittent fasting is also known as time-restricted eating, helps regulate the expression and activity of proteins and other cellular functions known to influence health and aging. In other words, it’s not so much of what you eat, but when you eat. The simple act of limiting food intake increases lifespan and reduces age-associated disorders such as diabetes and heart disease. This action may also boost our resistance to other diseases and ultimately help extend lifespan.
Sources:
Rafel de Cabo, PhD and Mark P. Mattson, PhD. Effects of Intermittent Fasting on Health, Aging, and Disease. The New England Journal of Medicine, December 26, 2019.
Benefits of Intermittent Fasting. Life Extension, February 2021
Diana Licalzi, MS, RD. How Intermittent Fasting Impacts Longevity: A Summary of the Research. October 23, 2020
Obesity is in the news (as usual) again this week with the estimate that by 2030, 42% of Americans will be obese (not just overweight, but obese). Compare that to our current U.S. rate of 36%. For months now nutrition “experts” are diligently trying to determine the cause of our national eating disorder and suggestions have been made smaller from portion sizes, too much fat, too many carbs, obesogens (chemicals in the food), processed and fast food, sugar-sweetened drinks, not enough sleep, not enough calcium, bacteria in the gut, and even viral infections. The list goes on and it still may be seen that all these factors may be contributory along with those still remaining to be studied.
The old long-held paradigm of calories in, calories out has been questioned. This advice works for some people, but for others it just doesn’t apply as we struggle with calorie restriction and increased exercise. The weight often creeps back even though some food and exercise habits have improved.
Studying food in different cultures allows us to look at the different ways people view food and how their diets affect their obesity and chronic disease rates. One culture stands out above all – the French, as they have a fairly high fat diet with low rates of obesity and heart disease. This is often called the “French Paradox”. How do they do it, we ask? The French, especially those residing in the northern and central regions, traditionally enjoyed cakes, pastries, and cheese that would make our arteries slam shut and pounds creep onto our waistlines.
So what are the facts about the traditional diets (around 10 years ago) of the French that kept them healthy and slim?
The French diet was high in saturated fat – 35-38% of total calories came from fat compared to around 34% in the U.S.
According to one study with French participants, only 14% derived less than 30% of their energy from fat, and only 4% derived less than 10% of their energy from saturated fat.
Their heart protective HDL-cholesterol and rates of hypertension were about the same as they are in North America, but the total serum cholesterol levels were higher.
They also smoked.
So tally these factors up and you would think that the French would have had at least a higher risk of heart attacks that North Americans. But the opposite was true.
According to the American Heart Association, out of thirty-five selected countries, France reported deaths from heart disease that were among the lowest in the world – second only to Japan.
In addition, they were better at combating cancer. For example, they reported incidence rates of breast cancer that were 50% lower, on average, than in the U.S. Also, their rates of colon and prostate cancers were roughly 30 and 60% lower, respectively, than those in the U.S.
Were they obese? Of course not. They were leaner – back in the late 1990’s, only 8% qualified as obese. How much of this is genetic? Probably not much since when the French moved to Montreal and began to consume a more U.S-style diet, “they gained weight and their heart disease rates began to resemble those of the U.S.
French Diet Secrets at a Glance:
Moderate drinking. The French have always mastered the art of moderate drinking. Their tempered one-to-two drinks-a-day habit may be what kept their hearts healthier despite their traditional high fat diet. And the French rarely drank alcohol without food. Think – no Happy Hours.
Lots of fruits and vegetables (no surprise). Even though the diet was high in fat, the French ate traditionally on average, four or more servings of vegetables a day.
No snacking. This is where the traditional French diet so differed from the U.S. typical dietary habits. Americans reported snacking on average three snacks a day contributing about 20% of day’s total calorie intake, the French did not usually partake of this between-meal ritual. Think of our supermarket aisles laden with snack foods.
They also did not eat pastries every day – they considered these as treats for special occasions. What’s for dessert? Mostly fruit after dinner.
A Megameal Lunch. A study showed that the French had consumed about 60% of their daily calories by 2:00 p.m. each day compared with an American group taking in only 40% of their calories by 2:00 p.m. then having a snack a couple of hours later and ate the largest meal at dinner.
Curtail Dieting. The French rarely take dieting to extremes. Prepackage meals and diet foods were considered an “insult” to their palates. Instead, they made small changes like limiting butter or cutting down on cheeses to lose extra pounds that may creep on.
More is Not Better. When the French visit the U.S. they are appalled at the quantity of food served. They find taking home leftovers or “doggy” bags comical. since they never serve you that much in the first place. If you overindulge, just cut down the next meal or day to compensate and constantly be aware of the amount served. So enjoy in Moderation.
The French have an attitude about food- they savor it. To quote Julia Child “If you’re afraid of butter, use cream.”
Fat is a key nutrient in our diet and is often the first thing you may note on a food label. Most foods contain a mixture of many different types of fat: the commonest are saturated, monounsaturated, polyunsaturated and trans fats. Polyunsaturated fats are divided into two major families: omega-6 and omega-3’s. Are some good and some bad.? This conundrum is often debated among nutritionists and still a definitive answer remains elusive.
What exactly are the omega-6 and omega 3 fats?
We have to begin with the polyunsaturated essential fatty acids, linoleic (omega-6) (LA) and alpha linolenic acids (omega-three) (ALA). They are called essential because they cannot be made in the body and must be acquired from the diet.
Linoleic acid (LA) is required for growth, healthy skin and normal functioning of the reproductive system and is a structural part of cell membranes. Foods high in omega 6 fats include unhealthy foods like processed snacks, fast foods, cakes, fatty meats, and cured meats. Other omega 6 foods are healthy including tofu, walnuts, and peanut butter. They are also prevalent in vegetable oils, like corn oil, safflower, sunflower and soybean oils. Linoleic acid is converted in the body to another fatty acid called arachidonic acid (AA). Food sources of AA include meat, poultry, and eggs. The adequate daily intake (AI) for omega 6 foods is 17000 mg per day.
Alpha linolenic acid (ALA) is also a structural compound of cell membranes and found in high amounts in the brain. Alpha linolenic acid is found in walnuts, dark, leafy green vegetables, flaxseed and chia seeds, canola and soybean oils.
ALA is converted in the body to two more fatty acids called eicosapentaenoic acid (EPA) and docosapentaenoic acid (DHA) which is what you find on fish oil supplement labels. This conversion rate of ALA to EPA can be slow and may depend on many factors, one being the ratio of omega-6 to omega-3 fatty acids.
EPA and DHA are found in fish, krill, and algae oil capsules as well as in fatty fish like salmon, tuna, sardines, mackerel, herring and trout. The AI for omega-3 fatty acids is 1.6 grams (men) and 1.1 g (women).
Arachidonic acid and EPAare necessary for making hormone-like compounds called eicosanoids, hormone-like compounds that participate in regulation of blood pressure, blood clotting, inflammation, and a host of other important body functions.
So, the major players so far are: LA, ALA, AA, EPA, and DHA.
What is the omega-6/omega-3 ratio?
It is not enough to consume adequate levels of omega-3 fats but to avoid over-consumption of omega-6 fatty acids. Most modern diets contain excessive amounts of omega-6s and insufficient amounts of omega-3s. Americans regularly eat vegetable oils but eat fish infrequently, so we end up with many more omega-6s and fewer omega-3s.
The optimal 6 to 3 ratio approaches 4:1 that may be difficult for some people in our current food environment to achieve, so we try for 4:1 in hopes of realistically attaining less than 10:1. On average in the U.S., the omega-6 to omega-3 ratio is a disastrous 16:1. Soybean oil, an omega-6 is so ubiquitous in the food supply that an astounding percent of calories from fat in the American diet (especially processed foods) are estimated to come from this single omega-6 source.
How Do Eicosanoids Affect Health?
Omega-6 fatty acids produce eicosanoids that tend to favor higher blood pressure, more blood clotting, and inflammatory compounds in the body. They are often referred to as “bad” eicosanoids.
Omega-3 fatty acids produce eicosanoids with opposing healthier effects, i.e., lower blood pressure, less blood clotting, and anti-inflammatory effects. They are often referred to a “good” eicosanoids.
Eicosanoids from omega-3 EPA can diminish the effects of the “bad” eicosanoids by producing opposing compounds that will help tip the ratio back to a more favorable eicosanoid environment in the cell.
Another way to improve the fatty acid ratio is to help block excess arachidonic acid formation. By making sure your body has an adequate amount of EPA that acts as an inhibitor of the enzyme that can produces the “bad” eicosanoids. The higher the EPA in the diet, the more the enzyme is inhibited, and the less “bad” eicosanoids are produced.
The problem with vegetable oils
“Vegetable oils that turn rancid easily have been used since lard was designated as having a high saturated fat content when the low-fat craze to prevent heart disease was in full swing. The troubled history of these oils has never been resolved. In a series of workshops in the 1980’s, it was observed that using diets high in soybean oil showed subjects dying of cancer at very high rates. Gallstones were also associated with diets high in vegetable oils. Subsequent research demonstrated that these oils that are high in omega-6, compete with the healthier omega-3’s found in fish virtually at important spots in every cell membrane throughout the body, including those in the brain.” (Nina Teicholz, The Big Fat Surprise).
The vast amount of omega-6 that has entered our food supply via vegetable oils appear to have literally swamped the omega-3’s (the supply of which has remained relatively constant over the past century. (Teicholz, page 275-6). Conversely, the American Heart Association encourages Americans to eat more vegetable oils due to their ability to lower LDL-cholesterol (the bad cholesterol.)
Nonetheless, excessive intake of polyunsaturated fatty acids, including omega-3 and omega-6, has several risks. The double bonds in the fatty acid molecules are very reactive. They tend to react with oxygen, forming chain reactions of free radicals. These free radicals can cause cell damage, which is one of the mechanisms behind aging and the onset of cancer.
If you want to improve your ratio of omega-6 to omega-3, it’s probably a bad idea to eat a lot of omega-3 to compensate. Having a relatively low, balanced amount of each is best. Using olive oil in salad dressings and coconut oil for cooking is recommended. Olive oil contains monounsaturated fat and coconut oil is more stable since it has more saturated fat content. Neither are part of the omega-6 or omega-3 families.
What to Take Away from all this:
Linoleic acid (LA), an omega-6 fatty acid, and α-linolenic acid (ALA), an omega-3 fatty acid, are considered essential fatty acids because they cannot be made in the body by humans.
Both omega-6 and omega-3 fatty acids are important structural components of cell membranes, serve as precursors to eicosanoids and provide a source of energy. Long-chain omega-3 polyunsaturated fatty acids in particular exert anti-inflammatory effects; it is recommended to increase their presence in the diet.
The long-chain omega-3 fatty acids, eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), can be synthesized from ALA, but due to low conversion efficiency, it is recommended to consume foods rich in EPA and DHA or consume fewer omega-6 foods.
Some but not all observational studies using supplements have found fish intake to be associated with lower risks of cognitive deterioration and Alzheimer’s disease, but it is not yet clear whether supplementation with marine-derived omega-3 PUFA can help prevent cognitive decline. There is a great need for intervention studies, especially with DHA to determine if improvements in brain health will occur. The Rancho Bernardo Study of Healthy Aging found a protective effect of DHA from diet on various aspects of cognitive decline and/or dementia.
Best to cut down on omega-6 foods (processed and junk foods), add a couple of fish meals a week, use olive oil for salads, coconut oil for cooking.
Top 10 Foods with the Highest Omega 3 to Omega 6 Ratio
Food
Ratio of Three to Six
Snow crab (3 oz)
61:1
Atlantic cod (6 oz)
29:1
Tuna (6 oz)
25 :1
Mussels (3 oz)
25:1
Broccoli Rabe (1 cup)
7:1
Spinach (1 cup)
5:1
Flax seeds (1 oz)
4:1
Mangos (1 cup)
3;1
Lettuce (1 cup)
2:1
Kidney beans (1 cup)
2:1
Sources:
Judith E. Brown. Nutrition Now Seventh Edition, 2013.
Have you ever wondered why some people gain weight very easily and others can eat all they want and not gain an ounce? Please read a previous post entitled “Born to Be Fat?” Click here.
Ethan Sims of the University of Vermont studied prisoners at a nearby state prison who volunteered to gain weight. They succeeded with great difficulty to increase their weights on average by 20-25% and it took 4-6 months.
Some consumed 10,000 calories/day and once fat, their metabolisms increased by 50% When the study ended, the prisoners had no trouble losing weight. Within months, they were back to normal and kept their weights stable.
These results suggest that there is a reason that overweight people cannot stay thin after they diet and that thin people cannot stay fat when forced to gain weight. The body’s metabolism speeds up or slows down to keep weight within a narrow range.
Recently, some new research adds more insights about how our DNA is involved. Here are some highlights:
Researchers deleted a gene called MRAP2 in mice that acts in the brain and controls how quickly calories are burned. It turned out that this gene helped another gene known to control appetite. These animals ate the same amount of calories as lean mice, but gained weight.
They also found a mutation in the same gene in a severely obese child and are now searching for other mutations that have the same or similar effects. If the gene that was helped to control appetite was not controlled by the helper gene (MRAP2), then the animals developed tremendous appetites, gained the weight without eating more calories and the weight resulted in fat accumulation.
People are often blamed for their own lack of self-control over eating or that they do not exercise, but in some people this is just not the case.
Researchers based at University College London reported that a specific form of a gene previously linked to obesity, FTO, could increase craving for high-fat foods. This gene makes an appetite hormone named ghrelin that works on the brain’s pleasure center to make high calorie foods more appetizing and desirable.
In this study, the researchers divided a group of 359 healthy men of normal weight by their FTO gene makeup. The majority of the men had low-risk versions of the gene, while 45 of the participants had mutations that have been linked to greater appetite and caloric consumption. They then measured levels of ghrelin both before and after meals that the participants ate. The men with the low-risk form of FTO showed a significant drop in ghrelin levels after the meals that signified that they were full while the men with the mutated form did not show this effect.
It is thought that about 70% of obesity causes are genetically controlled but that environmental influence on the genes can certainly occur.
The results did not mean that people are completely helpless to control their weight, but it does appear that people who tend to be overweight have a greater battle with their genes if they want to lose weight and maintain that weight loss.
As stated before, body weight is so highly regulated by so many physiological and psychological factors with many body systems involved, especially the brain. Maybe in the next few years or so, we can begin to figure out what actually causes weight gain and that it’s not just gluttony and lack of self-control. All these studies are valuable pieces of the puzzle.
Hooked on Food: A Battle in the Brain? The Reality of Food Addiction
Our brains maintain a healthy body weight by signaling when to eat and when to stop. Hormones regulate feeding circuits that control appetite and satiety, but fatty sugary foods can motivate some people to overeat. The more they have the more they want, a sensation common in drug addiction.
Sugar in the form of glucose provides the body with quick energy. But lately, we’ve gone way beyond the Call of Duty. 200 years ago, the average American ate about 2 pounds of sugar per year. Today we each eat about 152 pounds a year according to the US Department of Health and Human Services. This sharp increase in the consumption of sugar is no mystery. Sugar is cheap, plentiful, and it tastes great.
Some doctors and researchers classify sugar as an addictive drug because this refined white crystal triggers the pleasure and reward centers in our brain much like a drug does. It was already well established that sugar consumption will light up the nucleus accumbens and other areas of the brain that are collectively known as the reward centers, generating intense feelings of pleasure when we engage in acts like eating. Doctors are trying to curb our out-of-control sweets habit. The American Heart Association recommends that adult men consume no more than 38 grams or 9 teaspoons of sugar daily, women only 6 teaspoons, and children even less. The latest 2025 dietary guidelines recommends even smaller amounts for daily consumption: no more than 30 grams of added sugar a day for an adult male. However, these numbers fall far below what a typical American actually consumes. An average soda is 39 grams and a bowl of cereal is 20 grams and that’s without dumping more spoonfuls of sugar on top of it.
Is Sugar Addictive?
A recent study showed that rats can be addicted to foods, too. Actually, these foods sound very similar to those found in the Standard American Diet. The researchers gave rats unlimited access to standard chow as well as to a mini cafeteria full of appetizing high calorie foods: sausage, cheesecake, chocolate. The rats decreased their intake of the healthy but bland items and switched to eating the cafeteria food almost exclusively. They gained weight. They became obese.
The research then warned the rats as they were eating by flashing a light that they would receive a nasty foot shock. Rats eating the bland chow would quickly stop and scramble away, but time and again the obese rats continued to devour the rich food, ignoring the warning that they had been trained to fear. Their hedonic desire overruled their basic sense of self preservation.
“We now have the evidence for just how easy this is. People eating ultra processed, palatable foods are likely to eat more calories and no surprise, gain weight. We know this from a clinical trial run by Kevin Hall and his colleagues at the National Institutes of Health. They managed to convince 20 adults to live in a controlled metabolic ward for a month period. They gave the volunteers one of two diets unprocessed or ultra processed that were matched for calories, fats , carbohydrates, protein, and fiber. For two weeks the volunteers could eat as much as they wanted of the assigned diet; they then could eat as much as they wanted of the other diet for two weeks. The results were stunningly clear. On the ultra processed diet, the volunteers consumed many more calories – 500 — more a day than when eating the unprocessed diet. They also gained a pound week.
This experiment tells us that there is something about sweet, salty, and fatty foods that makes us want to eat more of them and to be unaware of how much more we’re eating. Salads and fruits do not trigger this kind of response.” Source: Marion Nestle. Let’s Ask Marion: What you need to know about the politics of food, nutrition, and health. California Study in Food and Culture. 2020.
Did they become “hooked on food”? An inability to suppress a behavior, despite the negative consequences, is common in addiction. Scientists are finding similar compulsiveness in certain people. Almost all obese individuals say they want to consume less, yet they consume or continue to overeat even though they know that doing so can have shockingly negative health or social consequences. Studies show that overeating juices up the reward systems in our brain so much so in some people that it overpowers the brain’s ability to tell them to stop eating when they have had enough. As with alcoholics and drug addicts, the more they eat the more they want. Whether or not overeating is technically an addiction if it stimulates the same brain circles as drug use in the same way, people also can possibly be “addicted to food.”
What to Do? Protein to the Rescue
Many peoples’ relationship with sugar typically starts when they wake up in the morning. Many start the day with a sweet bowl of cereal or a muffin (at 600 calories) for breakfast. But this pattern can set you up to fail, so many nutritionists recommend to focus more on protein.
Protein helps stabilize blood sugar which helps keep you out of fight or flight reactions and protein also provides the building blocks for your brain neurotransmitters including serotonin and dopamine. Many nutritionists advise their patients to eat protein such as eggs, cheese, nuts, peas, beans, and or even a protein shake at least an hour after they get up, and with every meal. If you snack before bed, make sure that it has protein too. Even if we strive to avoid sugar, it is often ubiquitous in our food culture appearing in many processed foods. If you have trouble saying no to sweets, it is recommended to eat protein proactively to keep temptations in check. This way you can help to avoid another binge on your favorite indulgence promoted by the American food industry and your brain, which is actually in on the hijacking, by the way.
FOOD, FACTS and FADS 